Basal PLSCR1 expression is highly induced in EBV-infected NPC cell line C666-1 and xenograft tumors C15 and C17, but is undetectable in EBV-positive B cells, suggesting its role in the switch from latent to lytic EBV infections (Kusano and Ikeda, 2019). Here, PLSCR1 is linked to nasopharyngeal carcinoma.