Previous studies have linked NUP62 to neurodegenerative disorders such as ALS and FTLD, demonstrating that the overexpression of NUP62 can effectively rescue eye degeneration in flies.30–32 However, the changes in NUP62 expression in human stem cells and its role as a regulator of senescence have yet to be thoroughly investigated. This evidence concerns the gene NUP62 and amyotrophic lateral sclerosis.