The results in Fig. S4A–4C showed that overexpression of CPT2 obviously attenuated the promotion of ferroptosis caused by SLC25A42 silencing in MKN-1 cells, while knockdown of CPT2 rescued the suppression of ferroptosis caused by forced SLC25A42 expression in SNU-638 cells, suggesting that CPT2 are required for the suppression of ferroptosis by SLC25A42 in GC cells. This evidence concerns the gene CPT2 and gastric cancer.