The sporadic, noninherited, wild-type form (wild-type transthyretin cardiac amyloidosis [wtATTR-CA]) primarily affects older, predominantly male individuals, whereas the hereditary form (hereditary transthyretin cardiac amyloidosis [hATTR-CA]) presents with a variable clinical phenotype, often involving both cardiomyopathy and polyneuropathy.1 Here, TTR is linked to cardiac amyloidosis.