AKT1 and neoplasm: The combination of ΒMSC-exos and SGC-7901 cells (subcutaneous tumor model of gastric cancer cell line) increased the ability of cancer cells to be transplanted, along with promoting a stronger proliferation potential through ERK 1/2 pathway activation, whereas the combination with cells of multiple myeloma led to enhanced survival via the p38, the c-Jun N-terminal kinase p53, and Akt pathway [57,58].