Especially in neuroinflammation, studies have reported a reduction in the production of cytokines, such as IL-1β, IL-6, TNF-α, IL-12, and IL-17, and chemokines, such as IFN-Ɣ, MCP-1, MIP-1a, and MIP-1b [43,44] in allergic asthma. This evidence concerns the gene CCL3 and allergic asthma.