The authors showed that the most important red blood cell polymorphism was heterozygous α+-thalassemia (37.8%), followed by G6PD A deficiency (16.4%), heterozygous sickle cell trait (15.9%), and G6PD A- deficiency (13.5%), and homozygous α+-thalassemia (5.2%) [30]. The gene discussed is G6PD; the disease is alpha thalassemia spectrum.