Within both COVID-19 cohorts, independent of the occurrence of thromboembolic outcome, inflammatory-related proteins (eg, leucine-rich α2-glycoprotein 1, S100A8, S100A9, CRP, and SAA1), von Willebrand factor (VWF) and neutrophil-related proteins (eg lactotransferrin, myeloperoxidase, and neutrophil defensin 1) were increased in abundance compared with healthy controls (Supplementary Figure S3, Supplementary Table S1G). This evidence concerns the gene LTF and COVID-19.