Activation of Jun, a transcription factor in the JNK pathway, promotes transcription of genes involved in many fibrotic diseases, including renal disease, and is associated with increased expression of CD47, an anti‐phagocytic protein that promotes fibrosis (Cui et al., 2020; Grynberg et al., 2017; Wernig et al., 2017). This evidence concerns the gene JUN and kidney disorder.