Platelet destruction in ITP is primarily mediated by autoantibodies targeting glycoproteins (GP) IIb/IIIa and GPIb/IX, leading to Fc gamma receptor (FcγR)‐dependent phagocytosis and complement‐mediated clearance in the spleen and liver [6, 7, 8]. The gene discussed is FCGR2A; the disease is autoimmune thrombocytopenic purpura.