NFKB1 and metabolic dysfunction-associated steatotic liver disease: The latest evidence pointed out that oxidative low-density lipoprotein (oxLDL) activated the NF-κB signaling pathway in double negative T (DNT) cells through CD36 receptors, upregulated the expression of HIF-1 α and ACSL4, induced ferroptosis in DNT cells, thereby disrupting liver immune homeostasis and accelerating the progression of MASLD to fibrosis and cirrhosis [31].