Other genes, such as TCF4 mutation, SLC4A11, Zinc finger E-box-binding homeobox 1 (ZEB1), lipoxygenase homology domains 1 (LOXHD1), and ATP/GTP binding protein-like 1 (AGBL1), are more closely associated with the late-onset form of FECD [63,64], with a trinucleotide repeat expansion in TCF4 being the most prevalent mutation [65]. The gene discussed is LOXHD1; the disease is Fuchs endothelial corneal dystrophy.