Although sequence analyses revealed notable homology between ATP13A3, ATP13A4 and ATP13A2,19 increased aberrant N6‐methyladenosine modification of ATP13A3 promotes colorectal tumorigenesis, whereas high ATP13A4 expression positively correlates with progression‐free survival in ovarian cancer patients.20, 21. This evidence concerns the gene ATP13A3 and ovarian carcinoma.