GFAP has been shown to increase in peripheral blood following neurological trauma and decrease over time,8 but GFAP has also been linked to mental health disorders and suicide in populations both exposed and unexposed to head trauma.9 Kulbe et al. 10 demonstrated in Transforming Research and Clinical Knowledge in Traumatic Brain Injury (TRACK-TBI) that lower GFAP concentrations on the day of mild TBI were associated with higher risk of developing post-traumatic stress disorder at 6 months post-injury. This evidence concerns the gene GFAP and acute stress disorder.