Furthermore, AKT-induced lncRNA VAL (vimentin-associated lncRNA, LINC01546) competes with the E3 ligase tripartite motif containing 16 (Trim16) for binding to vimentin, thereby inhibiting the ubiquitination and degradation of vimentin protein at the posttranslational level, which in turn promotes the metastasis of lung adenocarcinoma [26]. Here, VIM is linked to lung adenocarcinoma.