Moreover, other interventions aimed at normalizing excess H2S levels, for example by overexpression of SOD1, or by treatment of the cells with pleiotropic oxidized carbon nanozymes (which both catalytically oxidize H2S and dismutate superoxide radicals) also exert beneficial effects in DS cells due to their ability to reduce the "H2S burden" of these cells [29]. This evidence concerns the gene SOD1 and Dravet syndrome.