For example, eosinophils have been shown to play a protective role in limiting AAA progression by modulating macrophage and monocyte polarization.[36] Similarly, group 2 innate lymphoid cells contribute to AAA prevention in mice by promoting eosinophil activation through IL‐5 signaling.[37] These findings underscore the significance of immune cell interactions in AAA pathogenesis. Here, IL5 is linked to triple-A syndrome.