Since diet is the primary focus of GLUT1-DS treatment, as well as being of general interest for other diseases with suspected association with redox balance in the brain, the next step in our analysis was an attempt to restore normal levels of redox equivalents in the overall NGV (NAD+/NADH, ATP/ADP, and NADP+/NADPH) with various supplemental nutrient therapies (see S1 Text for procedure details). The gene discussed is SLC2A1; the disease is Dravet syndrome.