Accumulating evidence indicates that multiple CSC markers (e.g., ALDH and c-kit/CD117) and conserved signaling pathways (e.g., Wnt and Notch) are critically involved in OC pathogenesis, including tumor initiation, transcoelomic metastasis, hematogenous dissemination, and chemoresistance [20]. The gene discussed is KIT; the disease is neoplasm.