Previous studies have shown that c‐Myc can interact with the promoter of SREBP1, resulting in the reprogramming of fatty acid metabolism in liver cancer cells.[14] Moreover, c‐Myc has been demonstrated to increase the synthesis of particular eicosanoids in lung cancer cells, promoting the progression of lung cancer.[24b] In B‐cell lymphoma cell lines, c‐Myc stimulates increases in the mRNA and protein levels of crucial lipid synthesis enzymes, including ACACA, FASN, and SCD1. This evidence concerns the gene SREBF1 and lung carcinoma.