The study also found that neutrophil‐derived IL‐17A attracts more neutrophils by upregulating chemokines from keratinocytes via the IL‐17 signaling pathway in GPP skin, leading to an inflammatory crosstalk and neutrophil influx, therefore the inhibition of IL‐17A can be effective in GPP treatment.13 Here, IL17A is linked to psoriasis 14, pustular.