Given the inconclusive nature of the initial workup, further testing is warranted to investigate other potential causes of hypercoagulability, such as prothrombin G20210A mutation, methylenetetrahydrofolate reductase gene mutations, protein S deficiency, JAK2 V617F mutation, and antiphospholipid antibody testing. The gene discussed is PROS1; the disease is hyperinsulinemic hypoglycemia, familial, 4.