Furthermore, despite the fact that ATF4 itself was not deregulated by Ocoxin in all the analyzed cancer models, it arises as a central protein in the correlation among the genes altered by the compound (except for KIF20A, RAD54L and ESPL1) in common in COAD, PAAD, PRAD and TNBC. The gene discussed is RAD54L; the disease is prostate adenocarcinoma.