RAC3 and non-small cell lung carcinoma: This upregulation increases m6A modifications of Ras-related C3 botulinum toxin substrate 3 (RAC3) mRNA in NSCLC cells, enhancing the stability and translation of RAC3, activating the protein kinase B (AKT)/NF-κB pathway, and subsequently promoting NSCLC cell migration and invasion [64].