A previous study demonstrated that A-to-I RNA editing at position S367G in AZIN1 may confer a gain-of-function effect, promoting NSCLC tumorigenesis through inhibition of antizyme-1-mediated degradation of ornithine decarboxylase (ODC) and cyclin D1 (CCND1) oncoproteins [8]. Here, CCND1 is linked to non-small cell lung carcinoma.