It has been shown that proinflammatory cytokines released by immunological or tumour cells during CC, such as interleukin‐6 (IL‐6), tumour necrosis factor‐α (TNF‐α) and interleukin‐1 (IL‐1), cause muscle atrophy by activation of NF‐κB pathways [7]. The gene discussed is NFKB1; the disease is neoplasm.