Altogether suggests that CRS induces the endogenous release of 5‐HT that tonically activates both 5‐HT3 and 5‐HT1A receptors leading to a reduced tonic GABAergic inhibition in GCs, as has been observed after chronic stress (Holm et al. 2011; Lee et al. 2016). The gene discussed is HTR3A; the disease is congenital rubella syndrome.