A previous study showed that HDAC3 deacetylates and stabilizes GS through HDAC inhibitor screening (but this screening does not include HDAC6 inhibitors) and confirmed the result with genetic silencing in liver cancer cells.[33] Similarly, we observed that T247, an HDAC3‐selective inhibitor, reduces GS protein levels in HepG2 cells (Figure S3C, Supporting Information). The gene discussed is GLUL; the disease is liver cancer.