Hypocalcemic tetany occurs in the postnatal period due to the active transport of calcium from the mother to the foetus during gestation leading to secondary hypercalcaemia in the foetus, this process is interrupted after delivery and hypocalcemic tetany occur as a result of the inability of the mother’s PTH to pass through the placental membranes and the prolonged suppression of foetal parathyroid function, which prevents the newborn from mobilizing endogenous calcium reserves [6]. This evidence concerns the gene PTH and Hypercalcemia.