Conversely, the DC-like transition via enforced PU.1 or C/EBPβ expression, promoting antigen presentation in TIS as measured by a fluorogenic ovalbumin substrate (Supplementary Fig. 10c), rendered TIS-capable but not Suv39h1-deficient Eμ-myc;bcl2 lymphomas more susceptible to TTIS and/or Tnaïve killing, and resulted in higher cytotoxicity against lymphomas in TIS (Supplementary Fig. 10d). This evidence concerns the gene BCL2 and lymphoma.