Whole-genome sequencing analyses have demonstrated that compared to non-gestational tumors, epithelial cells from breast cancers diagnosed during pregnancy have higher expression of genes that regulate immune and inflammatory responses (e.g., PD1, PD-L1), DNA repair (e.g., BRCA1/2, FEN1, Sig20), cell proliferation (e.g., IGF1 and B-catenin), and pro-oncogenes (e.g., MYC, SRC, FOS) [6, 7]. The gene discussed is BRCA1; the disease is breast cancer.