MALAT1 regulates tumor-related inflammation and promotes the proliferation, motility, and epithelial–mesenchymal transition of tumor cells [24], whereas nuclear paraspeckle assembly transcript 1 (NEAT1) is involved in the cellular response to DNA damage and targets multiple pro-inflammatory factors, such as nuclear factor-kappa B (NF-κB) and cytokines (e.g., IL-1, IL-6, and TNF-α), thereby maintaining the inflammatory response [25,26]. The gene discussed is NFKB1; the disease is neoplasm.