These experimental findings bolster the results of genome-wide association studies (GWAS) linking TNIP1 with several hyperinflammatory and autoimmune disorders, e.g., psoriasis [16,17], psoriatic arthritis [18], and systemic lupus erythematosus (SLE) [19], as we have reviewed [2,20]. This evidence concerns the gene TNIP1 and psoriasis.