In a study using green-tea-derived exosomes as antagomiR carriers, significant results were obtained for the prevention of aortic dissection, thereby developing a form resistant to stable transport and enzyme elimination of antagomiR, leading to vascular remodeling through matrix metalloproteinase 9 (MMP9) and myocyte enhancer factor 2D (Mef2D) pathways [148]. This evidence concerns the gene MEF2D and dissection.