Many of the networks inferred to be derived from scRNA-seq represent well-characterized pathways associated with GBM, including those involved in the inflammatory response (e.g., type II interferon, and interleukin-1 to -4) [62,64], immune cell chemotaxis (e.g., CCL/CXCL, and colony-stimulating factors) [65], and angiogenesis (e.g., platelet-derived growth factor and vascular endothelial growth factor (VEGF)) [66,67]. This evidence concerns the gene VEGFA and glioblastoma.