APOB and hepatocellular carcinoma: The aberrant glycosylation of ApoB, activated by N-acetylglucosaminyl transferase III (GnT-III), can inhibit ApoB assembly and block the synthesis and secretion of very-low-density lipoprotein (VLDL), leading to triglyceride accumulation in the liver, aiding in the progression to hepatocellular carcinoma (HCC) [88].