We further confirmed this by analyzing the Beat AML database that contains 3 DEK::NUP214 patients and 448 AML patients diagnosed with other subtypes, where this pattern of upregulation was observed as well (EYA3, p = 0.005, PRDM2, p = 0.024, SESN1, p = 0.013, data not shown). This evidence concerns the gene SESN1 and acute myeloid leukemia.