During AKI, renal immune and intrinsic cells produce numerous pro‐inflammatory cytokines, namely IL‐6, IL‐8, IL‐1β, TNFα, and TNF‐γ, as well as anti‐inflammatory cytokines at the site of inflammation, such as IL‐4, IL‐10, IL‐13, and TGF‐β. Here, TGFB1 is linked to acute kidney injury.