CXCL10 binds to CXCR3‐B receptors on the surface of tumor cells and thus directly inhibits proliferation and shortens the survival time of tumor cells; binding to the CXCR3‐A receptor attenuates FAS (CD95)‐mediated cell apoptosis, tumor cells secrete CXCL10, and chemotactic CXCR3+ CD4/CD8/Treg cells invade the tumor tissue [53]. The gene discussed is FAS; the disease is neoplasm.