STING1 and STING-associated vasculopathy with onset in infancy: The cGAS‐STING pathway can be activated independently of any ligand solely through STING translocation to the Golgi, which can result from STING trafficking gene mutations, supporting the development of autoimmune diseases such as STING‐associated vasculopathy with onset infancy (SAVI) or COPA syndrome.[36]