During this cycle, DCs can be activated by tumor‐derived DNA, resulting in the activation of STING and due to the produced type I IFNs, DC maturation as well as accumulation increases.[13] The activation of STING further mediated the differentiation of naïve T cells into IFNγ‐producing type 1 T helper cells and IL‐9 producing TH9 T cells. This evidence concerns the gene STING1 and neoplasm.