Other thrombogenic factors, such as non‐criteria APS positive for anti‐CLIgM, anti‐CLβ2GP1 complex, LAC (dRVVT), LAC (aPTT), transiently positive anti‐PEIgG, anti‐PEIgM, or anti‐PS/PT antibodies, protein S deficiency, factor XII deficiency, and increased platelet aggregation, did not improve the live birth rates (Table 5). The gene discussed is PRB2; the disease is autoimmune polyendocrinopathy.