KRT88P and infection: Hepatitis B virus infection typically presents in two serological phases: an early infection phase characterized by positive Hepatitis B surface antigen (HBsAg+) and negative hepatitis B core antibody (HbcAb−), which may become chronic; and a resolving non-active infection phase, where HBsAg becomes negative, and hepatitis B core antibody (Anti-HBc) becomes positive (HBsAg−/Anti-HBc+) [2,6,7].