The molecule has shown several beneficial effects that can efficiently counteract ALS and ferroptosis targets; in this regard, Zhao et al. demonstrated the repression of ROS level after RV treatment in ALS mice and that RV, through the overexpression of PGC1α, improved motor performance and survival in a mouse model of ALS [189]. This evidence concerns the gene PPARGC1A and amyotrophic lateral sclerosis.