Olmes et al. demonstrated a correlation between the prevalence of M2a-like (CD206+/CD68+) and M2c-like (CD163+/CD68+) macrophages with tubular injury, glomerulosclerosis, crescent formation, and interstitial fibrosis/tubular atrophy, suggesting a contributory role in fibrogenesis [89]. The gene discussed is CD68; the disease is glomerulosclerosis.