In a study by Qu [110], IAV H9N2-infected human colorectal adenocarcinoma HT-29 cells, used as an in vitro model for IAV infection, exhibited elevated levels of tBid, as well as the increased activation of caspases-8 and -9, enzymes acting upstream and downstream of Bid cleavage. This evidence concerns the gene BID and colorectal adenocarcinoma.