Interestingly, recent studies have revealed the role of STING in KP infection, showing that the absence of STING promotes the clearance of KP, as determined by intranasal infection of STING−/− mice, which, at 24 h post-infection, showed a 58% reduction in bacterial load in the lungs compared with wild-type infected mice [69]. This evidence concerns the gene STING1 and keratosis pilaris.