Differences in the central obesity index, white blood cell count, platelet-to-lymphocyte ratio, and triglycerides between GHSR-1a (+) and GHSR-1a (−) patients suggest that GHSR-1a expression influences the accumulation of abdominal fat, the immune response, inflammation, and lipid metabolism, thereby potentially altering cardiovascular risk profiles. This evidence concerns the gene GHSR and Abdominal obesity.