Furthermore, persistent chromosomal segregation errors have been demonstrated to promote cell invasion and metastasis through a STING‐dependent mechanism.[14] Similarly, another study reported that the transfer of GAMR from tumor cells to astrocytes triggers the production of inflammatory cytokines and activates the signal transducer and activator of transcription 1 and NF‐κB pathways, ultimately leading to tumor growth and chemoresistance.[15] Conversely, the cGAS‐STING pathway has also played a key role in advancing anti‐tumor research. This evidence concerns the gene NFKB1 and neoplasm.