Vitamin D has been shown to stop the growth of keloid fibroblasts by stopping the production of extracellular matrix triggered by TGF‐β and increasing the production activity of matrix metalloproteinase and working as an anti‐inflammatory mediator, so the study's goal was to find out if intralesional and systemic vitamin D3 injections can help treat hypertrophic scars and keloids, and to find out if not getting enough vitamin D3 is linked to a higher risk of scarring. Here, TGFB1 is linked to keloid.