SSTR2 and neoplasm: Using mice expressing KrasG12D in pancreatic precursor cells (KC (55)), mice with monoallelic loss of Sstr2 (Sstr2+/–), and crossed KrasG12D/Sstr2+/- mice, this group in more mechanistic detail analysed the effects of Sstr2 loss on tumor growth in the course of Kras-induced PDAC development.